Estudio Origen Sexualidad De Simon Levay (Inglés)
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http://www.jstor.orggrowth, nudear segregation,DNA repair,and meiosis, and deletion of HRR25 resultsin cell cycle defects.These phenotypes, coupled with the similarityof the HRR25 sequence to the sequenceof the Rafc-Mos protein kinasesubgroup (Fig. 3A), suggest that HRR25 might play a similarrole in S. cerevisiae growth and development. The defects in DNA doublestrandbreakrepairand aberrant growth propertiesrevealed by mutations in the HRR25 kinaseextend the possible functions of protein kinasesin cell growth and place HRR25 with CDC7 in a functionalcategoryof yeast kinase associatedwith DNA metabolism.
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