Teoria Celular
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Publicado: 6 de febrero de 2013
teoria celular
http://148.216.10.83/biologiaQFB1/teoria_celular.htm
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repression of E2F-4., J Biol Chem. 277(30):26741-52 (2002)
[40] J. William Harbour, and Douglas C.Dean Vol. 14, No. 19, pp. 2393-2409, (2000)
http://www.genesdev.org/cgi/content/full/14/19/2393 [consulta: 21 de nero del 2003]
[41] Paul A. Wade, Transcriptional control at regulatory checkpoints byhistone deacetylases: molecular connections
between cancer and chromatin, Human Molecular Genetics Vol. 10, No. 7 693-698 (2001) [en línea]
http://hmg.oupjournals.org/cgi/content/full/10/7/693 ,[Consulta: 28 de enero del 2003]
[42] Lundberg, A. S. & Weinberg, R. A. Functional inactivation of the retinoblastoma protein requires sequential
modification by at least two distinct cyclin-CDKcomplexes. Mol. Cell. Biol. 18, 753-761 (1998). /PubMed
[43] EP300: E1A binding protein (p300),NCBI, http://www.ncbi.nlm.nih.gov/LocusLink/LocRpt.cgi?l=2033, [en línea]
[consulta: 28 de enero del 2003][44] Chan, H. M., Krstic-Demonacos, M., Smith, L., Demonacos, C. & La Thangue, N. B. Acetylation control of the
retinoblastoma tumour-suppressor protein. Nature Cell Biol. 3, 667-674 (2001). /Article/PubMed
[45] Malumbres, M. & Pellicer, A. Ras pathways to cell cycle control and cell transformation. Front. Biosci. 3, D887-D912
(1998).[en línea]http://www.bioscience.org/1998/v3/d/malumbre/list.htm, [consulta: 27 de enero del 2003]
[46] Sicinski, P. et al. Cyclin D1 provides a link between development and oncogenesis in the retina and breast. Cell 82,
621-630 (1995). /PubMed
[47]Angeliki Malliri, Rob A. Van Der Kammen, Kristopher Clark, Maarten Van Der Valk, Mice deficient in the Rac activator
Tiam1 are resistant to Ras-induced skin tumours, Nature 417, 867 - 871 (2002);
[48]CCND1: cyclin D1 (PRAD1: parathyroid adenomatosis 1),NCBI
http://www.ncbi.nlm.nih.gov/LocusLink/LocRpt.cgi?l=893, [consulta: 28 de enero del 2003]
[49] Charles J. Sherr, The Pezcoller Lecture: Cancer...
http://148.216.10.83/biologiaQFB1/teoria_celular.htm
12 of 12
18/10/06 16:26
repression of E2F-4., J Biol Chem. 277(30):26741-52 (2002)
[40] J. William Harbour, and Douglas C.Dean Vol. 14, No. 19, pp. 2393-2409, (2000)
http://www.genesdev.org/cgi/content/full/14/19/2393 [consulta: 21 de nero del 2003]
[41] Paul A. Wade, Transcriptional control at regulatory checkpoints byhistone deacetylases: molecular connections
between cancer and chromatin, Human Molecular Genetics Vol. 10, No. 7 693-698 (2001) [en línea]
http://hmg.oupjournals.org/cgi/content/full/10/7/693 ,[Consulta: 28 de enero del 2003]
[42] Lundberg, A. S. & Weinberg, R. A. Functional inactivation of the retinoblastoma protein requires sequential
modification by at least two distinct cyclin-CDKcomplexes. Mol. Cell. Biol. 18, 753-761 (1998). /PubMed
[43] EP300: E1A binding protein (p300),NCBI, http://www.ncbi.nlm.nih.gov/LocusLink/LocRpt.cgi?l=2033, [en línea]
[consulta: 28 de enero del 2003][44] Chan, H. M., Krstic-Demonacos, M., Smith, L., Demonacos, C. & La Thangue, N. B. Acetylation control of the
retinoblastoma tumour-suppressor protein. Nature Cell Biol. 3, 667-674 (2001). /Article/PubMed
[45] Malumbres, M. & Pellicer, A. Ras pathways to cell cycle control and cell transformation. Front. Biosci. 3, D887-D912
(1998).[en línea]http://www.bioscience.org/1998/v3/d/malumbre/list.htm, [consulta: 27 de enero del 2003]
[46] Sicinski, P. et al. Cyclin D1 provides a link between development and oncogenesis in the retina and breast. Cell 82,
621-630 (1995). /PubMed
[47]Angeliki Malliri, Rob A. Van Der Kammen, Kristopher Clark, Maarten Van Der Valk, Mice deficient in the Rac activator
Tiam1 are resistant to Ras-induced skin tumours, Nature 417, 867 - 871 (2002);
[48]CCND1: cyclin D1 (PRAD1: parathyroid adenomatosis 1),NCBI
http://www.ncbi.nlm.nih.gov/LocusLink/LocRpt.cgi?l=893, [consulta: 28 de enero del 2003]
[49] Charles J. Sherr, The Pezcoller Lecture: Cancer...
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